Hormone Pellet Therapy in Oakland Park

Are you one of the many people in Oakland Park who are burning the candle at both ends and maybe only getting 4 or 5 hours of sleep a night? Are you also one of those guys having problems with his sex drive and feeling out of sorts? Well, recent studies done in Oakland Park in the last 3 years show that these symptoms could all be due to the effect of sleep on testosterone – just how, though, may be a chicken and egg question!

While it’s true that lower testosterone levels can be the cause of a sluggish sex drive and irritability it seems to be a matter of research opinion whether low sleep levels cause low testosterone or low testosterone causes lack of sleep.

Natural Support to Increase Level of Testosterone

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There's a growing interest in testosterone hormone replacement for treating symptoms related to aging. You've probably seen advertisements of virile, muscle bound men in their 60's and 70's.

Along with the growing interest there's also a growing amount of information. But much of it is anecdotal stories, misleading data and flat out, unproven myths. Especially as it relates to testosterone replacement therapy for women.

The fact is that medically administered, testosterone therapy is also used to successfully treat symptoms of hormone deficiency in pre and postmenopausal women. And two physicians-Dr. Rebecca Glaser and Dr. Constantine Dimitrakakis-are dispelling the misinformation about it through scientific research.

Dr. Glaser and Dr. Dimitrakakis focus on subcutaneously implanted, bio-identical hormones (human identical molecule) and not oral, synthetic androgens or anabolic steroids.

With that in mind, here are the 10 myths of testosterone replacement therapy for women.

Myth #1: Testosterone is a "male" hormone

Although men have a higher circulating level of testosterone than women, from a biological perspective, men and women are genetically similar. Both sexes include functional estrogen and androgen (testosterone) receptors. And while estrogen is popularly considered the primary female hormone, throughout a woman's lifespan, testosterone is actually the most abundant, biologically active hormone with significantly higher levels than estradiol. And as early as 1937, testosterone therapy was reported to effectively treat symptoms of the menopause.

Myth #2: Its only role in women is sex drive and libido

There's a lot of hype about testosterone's role in sexual function. But in reality, it's a fraction of the overall physiologic effect testosterone plays in women. That's because testosterone governs the health of almost all tissues including the breast, heart, blood vessels, gastrointestinal tract, lung, brain, spinal cord, peripheral nerves, bladder, uterus, ovaries, endocrine glands, vaginal tissue, skin, bone, bone marrow, synovium, muscle and adipose tissue.

The function of these tissues declines as testosterone declines. The result of this deficiency in both men and women includes dysphoric mood (anxiety, irritability, depression), lack of well-being, physical fatigue, bone loss, muscle loss, changes in cognition, memory loss, insomnia, hot flashes, rheumatoid complaints, pain, breast pain, urinary complaints, incontinence as well as sexual dysfunction. And just like for men, these symptoms are successfully treated in women through testosterone therapy.

Myth #3: It masculinizes females

Testosterone therapy has been safely and successfully administered in women for over 76 years. Rather than decrease a woman's femininity it increases it. Testosterone stimulates ovulation, increases fertility and safely treats the nausea of early pregnancy without adverse effects.

Sure, large doses of supra-pharmacological synthetic testosterone are used to treat female to male transgender patients to increase male traits like body hair. But this requires high doses over an extended period of time. Even then, true masculinization is still not possible. And these effects are reversible by simply lowering the dosage.

Myth #4: It causes hoarseness and voice changes

Hoarseness is most commonly caused by inflammation due to allergies, infectious or chemical laryngitis, reflux esophagitis, voice over-use, mucosal tears, medications and vocal cord polyps. Testosterone possesses anti-inflammatory properties. There is no evidence that testosterone causes hoarseness and there is no physiological mechanism that allows testosterone to do so.

Although a few anecdotal case reports and small questionnaire studies have reported an association between 400 and 800 mg/d of danazol and self-reported, subjective voice 'changes' an objective study demonstrated the opposite.

Twenty-four patients received 600 mg of danazol (synthetic testosterone) therapy daily and were studied for 3 and 6 months. There were no vocal changes that could be attributed to the androgenic properties of danazol. These conclusions are consistent with a one year study examining voice changes on pharmaco-logic doses of subcutaneous testosterone implant therapy in women by Glaser and Dimitrakakis.

Myth #5: It causes hair loss

Hair loss is a complicated, genetically determined process and there is no evidence that either testosterone or testosterone therapy cause it. In fact, from a medical perspective, dihy-drotestosterone (DHT), not testosterone, is considered the active androgen in male pattern balding.

There are many factors associated with hair loss. For example, it's common in both women and men with insulin resistance. Insulin resistance increases 5-alpha reductase, which increases conversion of testosterone to dihy-drotestosterone in the hair follicle.

In addition, obesity, age, alcohol, medications and sedentary lifestyle increase aromatase activity, which lowers testosterone and raises estradiol. Increased DHT, lowered testosterone, and elevated estradiol levels can contribute to hair loss in genetically predisposed men and women. But so can medications, stress and nutritional deficiencies.

In studies conducted by Glaser and Dimitrakakis, two thirds of women treated with subcutaneous testosterone implants have scalp hair re-growth on therapy. Women who did not re-grow hair were more likely to be hypo or hyperthyroid, iron deficient or have elevated body mass index. And none of the 285 patients treated for up to 56 months with subcutaneous T therapy complained of hair loss.

Myth #6: It has adverse effects on the heart

On the contrary, there is overwhelming biological and clinical evidence that testosterone promotes a healthy heart. Testosterone has a beneficial effect on lean body mass, glucose metabolism and lipid profiles in men and women. It is successfully used to treat and prevent cardiovascular disease and diabetes.

Testosterone also widens blood vessels in both sexes, has immune-modulating properties that inhibit plaque and strengthens the cardiac muscle. It improves functional capacity, insulin resistance and muscle strength in both men and women with congestive heart failure.

Myth #7: It causes liver damage

High doses of oral, synthetic androgens (e.g., methyl-testosterone) pass through the digestive system, are absorbed into the entero-hepatic circulation and can adversely affect the liver. But subcutaneous implants and topical patches avoid the entero-hepatic circulation and bypass the liver. So there is no adverse effect on the liver, liver enzymes or clotting factors.

Furthermore, non-oral testosterone does not increase the risk of deep venous thrombosis or pulmonary embolism like oral estrogens, androgens and synthetic progestins. And despite the concern over liver toxicities with anabolic steroids and oral synthetic androgens, there are only 3 reports of hepa-tocellular carcinoma in men treated with high doses of oral synthetic methyl testosterone. Even the report of benign tumors (adenomas) with oral androgen therapy is exceedingly rare.

Myth #8: It causes aggression

Although anabolic steroids can increase aggression and rage, this does not occur with testosterone therapy. Even supra-pharmacologic doses of intramuscular testosterone undecanoate do not increase aggressive behavior. But as stated before, testosterone can aromatize to estradiol. And there is considerable evidence among species, that estrogens, not testosterone, play a major role in aggression and hostility.

However, in studies conducted by Glaser and Dimitrakakis, over 90% of women treated with subcutaneous testosterone therapy have documented decreased aggression, irritability and anxiety. And this is not a new finding. Androgen therapy has been used to treat PMS for over 60 years.

Myth #9: It may increase the risk of breast cancer

It was recognized as early as 1937 that breast cancer was an estrogen sensitive cancer and that testosterone acted as a counter balance to estrogen. Clinical trials in primates and humans have confirmed that testosterone has a beneficial effect on breast tissue by decreasing breast proliferation and preventing stimulation from estradiol.

However, some epidemiological studies have reported an association between elevated androgens and breast cancer. But these studies suffer from methodological limitations, and more importantly, do not account for associated elevated estradiol levels and increased body mass index. And the cause and effect interpretation of these studies conflicts with the known biological effect of testosterone.

Although testosterone is breast protective, it can aromatize to estradiol and have a secondary, stimulatory effect on the estrogen receptor. But when testosterone is combined with an aromatase inhibitor in a subcutaneous implant, it blocks testosterone from aromatizing.

This form of treatment has been shown to effectively treat androgen deficiency symptoms in breast cancer survivors and is currently being evaluated in a U.S. national cancer study. In addition, Dimitrakakis and Glaser see a reduced incidence of breast cancer in women treated with testosterone or testosterone with anastrozole implants.

Myth #10: The safety of testosterone use in women has not been established

Testosterone implants have been used safely in women since 1938. Any real concerns would be well established by now.

Long-term data exists on the successful and safe use of testosterone in doses of up to 225 mg in up to 40 years of therapy. In addition, long term follow up studies on supra-pharmacologic doses used to 'female to male' transgender patients report no increase in mortality, breast cancer, vascular disease or other major health problems.

Many of the side effects and safety concerns attributed to testosterone are from oral formulations, or are secondary to increased aromatase activity due to elevated estradiol. This effect increases with age, obesity, alcohol intake, insulin resistance, breast cancer, medications, drugs, processed diet and sedentary lifestyle. Although often overlooked or not addressed in clinical studies, monitoring aromatase activity and symptoms of elevated estradiol is critical to the safe use of testosterone in both sexes.

Adequate testosterone is essential for physical, mental and emotional health in both sexes. Abandoning myths, misconceptions and unfounded concerns about testosterone and testosterone therapy in women allows physicians to provide evidence based recommendations and appropriate therapy

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Marijuana attacks your precious testosterone in almost every negative way possible. One study after another has shown that cannabis lowers testosterone. For example one research team found that "a reanalysis of existing data established that testosterone levels are depressed both after smoking one marijuana cigarette and after intravenous infusion of delta-9-tetrahydrocannabinol, a pharmacologically active component of marijuana". The same study concluded that it would take at least 24 hours for testosterone levels to normalize after marijuana use. (NOTE: It's not just the smoke - an IV will do it.)

Another study found that not only was testosterone decreased after short term marijuana use, but leutenizing and follicle stimulating hormone were lowered as well. And just to add to the endocrinological misery, the arch-villain and stress hormone cortisol was raised as well. There are also studies in animals and humans that strongly indicate that marijuana blunts growth hormone response as well. And so it is no wonder that animal studies show that marijuana use shrinks the testes. So, if you're not happy with lowered testosterone, infertility and elevated cortisol, you can sit around enjoying the fact that you've got a little more air flow through your boxers.

You should also know that there are many reports that chronic marijuana use leads to gynecomastia, i.e. "enlarged male breasts", due to its abundant amounts of phytoestrogens. One journal writer pointed out that "given the effects of marijuana on the HPG axis in males and the possibility that noncannabinoid components of marijuana smoke have affinity to the estrogen receptor, an association with gynecomastia is plausible but has not been convincingly demonstrated". Remember that estrogen fights against testeosterone in the body as well.

Marijuana has also recently been flagged as particularly dangerous for young people because it decreases seratonin and increases norepineprine. While these are not sex hormones like testosterone, these can alter mood negatively and, through prolonged use, may permanently alter anxiety levels and reaction to stress. Again, the researchers are suggesting this may have long term, possibly lifetime anxiety and mood repercussions. I would also add that any increase in stress will also likely lower testosterone as well.

So we ask the question, "Could someone please explain again why anyone in their right mind would smoke marijuana?" The only thing we can think of is the extra hydrogen cyanide. That's right - marijuana tobaco is much higher in hydrogen cyanide - probably five times higher - than cigarette tobacco. Maybe that partially explains why habitual pot smoking is so hard on the lungs and why cannabis use has also now been linked to the most aggressive form of testicular cancer.

Not to make the bad news even worse, but there is also considerable reported evidence of erectile dysfunction among chronic marijuana users. This is undoubtedly partially due to the lowered testosterone. However, the other reason was discovered by one study that showed marijauna effected Nitric Oxide and summarized by saying, "We conclude that early endothelial damage may be induced by chronic cannabis use (and endocannabinoid system activation". Let me translate that: it may take your sex life with it. If so, decreased sexual activity is also associated with lowered testosterone levels as well.

The tragedy with marijuana is that many cultures and youth are embracing marijuana as more "natural", but this is far from being the case. One recent study found that marijuana induces just as much cell toxicity and DNA damage as cigarette smoke. The researchers were very clear that marijuana displayed just as much cancer causing power as the cigarette smoke: "In addition, when corrected for total particulate matter yield, little difference was observed in the mutagenic activity of samples smoked under the extreme vs the standard regime for both tobacco and marijuana condensates".

In summary, there is significant evidence that marijuana lowers testosterone, nitric oxide, leutinizing hormone, growth hormone and raises cortisol at the same time. Hormonally, there is no justifiable reason for cannabis use.

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What is the underlying cause of impotence, depression, fatigue, excess body fat and osteoporosis in an estimated four million American men? Low Testosterone.

Natural supplements can be an alternative to creams, gels and patches. Dietary changes are slower but have less side effects.

For men, testosterone and DHEA ( a precursor hormone for testosterone) diminish after the age of 40. Actually the peak age is 17 and then production slowly falls off for the rest of your life. It does not become noticeable until around 40 plus.

Your doctor can perform a simple test to measure your testosterone. Normal levels range from 300 to 1,000 ng/dl.

Talk to him - you may be able to get some changes going using what nature has provided.

Traditionally Asia's most prized herb for hundreds of years is Ginseng root. Most of North America's crop of ginseng is mainly shipped to China. Ginseng is supposed to increase blood flow.

Sarsaparill contains a testosterone-like substance. Most main stream physicians will tell you that it has no effect.

Saw Palmetto at 120-360 mg daily is supposed to reduce the conversion of testosterone to estrogen. (see Low Testosterone)

Diet and Testosterone

Adjust your diet to make sure you get the good stuff. Zinc, Manganese and Niacin (B3) are absolutely essential. Add pumpkin seeds or sunflower seeds.

Milk Thistle is a good source of zinc and is very helpful to your liver.

Niacin is found in beef liver and brewer's yeast. If you go the beef liver route be sure it is grass fed beef. Use caution in supplements as Niacin (B3) in amounts over 500 mg may cause liver damage.

Of course, if you already have diabetes, glaucoma, gout, ulcers or any liver disease you must consult your physician before adding additional B3 supplements to your diet.

The FDA and traditionally physicians do not believe that DHEA supplements taken orally do any good. That being said, the suggested way to take DHEA is 2 weeks, discontinue for 2 weeks and then repeat. Taking this supplement daily continually is detrimental.

If you have read about Yohimbe and are tempted - use caution. This herb has been associated with panic attacks, hallucinations, elevated blood pressure, headaches and dizziness. It is also bad for the kidneys.

Flavonoids (whole grains, legumes, fruits, and vegetables) are protective in coronary heart disease, stroke and cancer. Research is being done to determine if one flavonoid, chrysin, found in high concentrations in honey could inhibit the aromatase action that turns testosterone into estrogen. If it does work, that would increase the level of testosterone. If it doesn't work, at least you are doing good things for your heart.

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Oakland Park, Florida

Oakland Park, officially the City of Oakland Park, is a city in Broward County, Florida, United States. As of the 2010 United States Census, the city's population was 41,363,[11] mainly due to annexation of North Andrews Gardens and Twin Lakes South. It is part of the Miami–Fort Lauderdale–Pompano Beach Metropolitan Statistical Area, which was home to 5,564,635 people at the 2010 census.

Originally named "Floranada" (a blend of Florida and Canada), the town was forced into bankruptcy after the hurricane of 1926. When the municipality reincorporated, residents chose to make it a city and voted for the name "Oakland Park." The original boundaries went from the Atlantic Ocean, west to what is now U.S. 441, and from the north fork of Middle River north to Cypress Creek Boulevard, but when the boundaries were reestablished, it was to approximately the west side of U.S. 1, west to Northeast 3rd Avenue and the north fork of Middle River north to what is now Prospect Road. Over time, it has expanded to its current boundaries, mainly due to acquiring a few other areas, such as recent annexations of previously unincorporated neighborhoods of Twin Lakes South and North Andrews Gardens.[12]

Oakland Park is located at 26°10′35″N 80°8′40″W / 26.17639°N 80.14444°W / 26.17639; -80.14444 (26.176362, -80.144509).[13] According to the United States Census Bureau, the city has a total area of 8.1 square miles (21.1 km2), of which 7.5 square miles (19.3 km2) is land and 0.69 square miles (1.8 km2) (8.40%) is water.[11]


Hormone Replacement Therapy For Men in Broward